Spremljaj
Matthew Pamenter
Naslov
Navedeno
Navedeno
Leto
Time domains of the hypoxic ventilatory response and their molecular basis
ME Pamenter, FL Powell
Comprehensive Physiology 6 (3), 1345-1385, 2016
1562016
Adaptive responses of vertebrate neurons to anoxia—Matching supply to demand
LT Buck, ME Pamenter
Respiratory physiology & neurobiology 154 (1-2), 226-240, 2006
1012006
The exceptional longevity of the naked mole‐rat may be explained by mitochondrial antioxidant defenses
D Munro, C Baldy, ME Pamenter, JR Treberg
Aging Cell 18 (3), e12916, 2019
982019
Hypoxia induces Kv channel current inhibition by increased NADPH oxidase-derived reactive oxygen species
M Mittal, XQ Gu, O Pak, ME Pamenter, D Haag, DB Fuchs, RT Schermuly, ...
Free Radical Biology and Medicine 52 (6), 1033-1042, 2012
932012
The naked truth: a comprehensive clarification and classification of current ‘myths’ in naked mole‐rat biology
R Buffenstein, V Amoroso, B Andziak, S Avdieiev, J Azpurua, AJ Barker, ...
Biological Reviews 97 (1), 115-140, 2022
902022
Cross-species insights into genomic adaptations to hypoxia
ME Pamenter, JE Hall, Y Tanabe, TS Simonson
Frontiers in Genetics 11, 743, 2020
832020
Endogenous GABAA and GABAB receptor-mediated electrical suppression is critical to neuronal anoxia tolerance
ME Pamenter, DW Hogg, J Ormond, DS Shin, MA Woodin, LT Buck
Proceedings of the National Academy of Sciences 108 (27), 11274-11279, 2011
832011
Anoxia-induced changes in reactive oxygen species and cyclic nucleotides in the painted turtle
ME Pamenter, MD Richards, LT Buck
Journal of Comparative Physiology B 177, 473-481, 2007
762007
Mitochondrial ATP‐sensitive K+ channels regulate NMDAR activity in the cortex of the anoxic western painted turtle
ME Pamenter, DSH Shin, M Cooray, LT Buck
The Journal of physiology 586 (4), 1043-1058, 2008
742008
Oxygen in demand: how oxygen has shaped vertebrate physiology
YA Dzal, SEM Jenkin, SL Lague, MN Reichert, JM York, ME Pamenter
Comparative Biochemistry and Physiology Part A: Molecular & Integrative …, 2015
722015
Mitochondria: a multimodal hub of hypoxia tolerance
ME Pamenter
Canadian journal of zoology 92 (7), 569-589, 2014
722014
Do naked mole rats accumulate a metabolic acidosis or an oxygen debt in severe hypoxia?
ME Pamenter, YA Dzal, WA Thompson, WK Milsom
Journal of Experimental Biology 222 (3), jeb191197, 2019
682019
Piscine insights into comparisons of anoxia tolerance, ammonia toxicity, stroke and hepatic encephalopathy
PJ Walsh, CM Veauvy, MD McDonald, ME Pamenter, LT Buck, MP Wilkie
Comparative Biochemistry and Physiology Part A: Molecular & Integrative …, 2007
682007
Naked mole rat brain mitochondria electron transport system flux and H+ leak are reduced during acute hypoxia
ME Pamenter, GY Lau, JG Richards, WK Milsom
Journal of Experimental Biology 221 (4), jeb171397, 2018
672018
Autophagy and apoptosis are differentially induced in neurons and astrocytes treated with an in vitro mimic of the ischemic penumbra
ME Pamenter, GA Perkins, AK McGinness, XQ Gu, MH Ellisman, ...
PLoS One 7 (12), e51469, 2012
652012
The hypoxia-tolerant vertebrate brain: arresting synaptic activity
LT Buck, ME Pamenter
Comparative Biochemistry and Physiology Part B: Biochemistry and Molecular …, 2018
642018
Behavioural responses of naked mole rats to acute hypoxia and anoxia
AN Ilacqua, AM Kirby, ME Pamenter
Biology letters 13 (12), 20170545, 2017
622017
AMPA receptors undergo channel arrest in the anoxic turtle cortex
ME Pamenter, DSH Shin, LT Buck
American Journal of Physiology-Regulatory, Integrative and Comparative …, 2008
602008
Adenosine receptors mediate the hypoxic ventilatory response but not the hypoxic metabolic response in the naked mole rat during acute hypoxia
ME Pamenter, YA Dzal, WK Milsom
Proceedings of the Royal Society B: Biological Sciences 282 (1800), 20141722, 2015
582015
Evidence of anoxia-induced channel arrest in the brain of the goldfish (Carassius auratus)
MP Wilkie, ME Pamenter, S Alkabie, D Carapic, DSH Shin, LT Buck
Comparative Biochemistry and Physiology Part C: Toxicology & Pharmacology …, 2008
582008
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