Longstanding racial/ethnic inequalities in morbidity and mortality persist in the United States.
Although the determinants of health inequalities are complex, social and structural factors …

In utero exposure to environmental stress in both animals and humans could result in long-
term epigenome alterations which further lead to consequences for adaptation and …

Mental disorders are highly complex and multifactorial in origin, comprising an elaborate
interplay of genetic and environmental factors. Epigenetic mechanisms such as DNA …

Fetal glucocorticoid overexposure is a key mechanism linking early development with later-
life disease. In humans, low birth weight associates with increased fasting cortisol …

Maternal prenatal stress has been linked to a variety of infant postnatal outcomes, partially
through alterations in fetal HPA axis functioning; yet the underlying pathobiology remains …

Aim: Sex-based differences in response to adverse prenatal environments and infant
outcomes have been observed, yet the underlying mechanisms for this are unclear. The …

The glucocorticoid receptor gene (NR3C1) is a critical component of the stress response
system. Cytosine methylation of NR3C1 has been repeatedly associated with trauma and …

This review summarizes current knowledge and outlines future directions relevant to
questions concerning environmental epigenetics and the processes that contribute to …

The integration of behavioral epigenetics' principles (eg, DNA methylation) into the study of
human infants' development has mainly focused on the effects of early adverse exposures …

Psychiatric disorders are clinically heterogeneous and debilitating chronic diseases
resulting from a complex interplay between gene variants and environmental factors …