Abstract Calcium–calmodulin (CaM)-dependent protein kinase II (CaMKII) is the most
abundant protein in excitatory synapses and is central to synaptic plasticity, learning and …

Summary The Ca 2+/calmodulin (CaM)-dependent protein kinase II (CaMKII) was touted as
a memory molecule, even before its involvement in long-term potentiation (LTP) was shown …

Long-term potentiation (LTP) of synaptic transmission is considered to be a cellular
counterpart of learning and memory. Activation of postsynaptic NMDA type glutamate …

Calcium/calmodulin-stimulated protein kinase II (CaMKII) is a family of broad substrate
specificity serine (Ser)/threonine (Thr) protein kinases widely expressed in many tissues that …

Abstract The Ca2+/calmodulin (CaM)-dependent protein kinase II (CaMKII) plays a
fundamental role in learning and possibly also in memory. However, current mechanistic …

Gap junctions establish direct pathways for cell-to-cell communication through the assembly
of twelve connexin subunits that form intercellular channels connecting neighbouring cells …

Summary The Ca 2+/calmodulin (CaM)-dependent protein kinase II (CaMKII) is a ubiquitous
mediator of cellular Ca 2+ signals with both enzymatic and structural functions. Here, we …

The reduced sleep duration previously observed in Camk2b knockout mice revealed a role
for Ca2+/calmodulin-dependent protein kinase II (CaMKII) β as a sleep-promoting kinase …

Cardiovascular disease is the major cause of death worldwide. The success of medication
and other preventive measures introduced in the last century have not yet halted the …

Higher brain functions are thought to require synaptic frequency decoding that can lead to
long-term potentiation (LTP) or depression (LTD). We show that the LTP versus LTD …