Beyond GWASs: illuminating the dark road from association to function

SL Edwards, J Beesley, JD French… - The American Journal of …, 2013 - cell.com
Genome-wide association studies (GWASs) have enabled the discovery of common genetic
variation contributing to normal and pathological traits and clinical drug responses, but …

Enhancer variants: evaluating functions in common disease

O Corradin, PC Scacheri - Genome medicine, 2014 - Springer
Gene enhancer elements are noncoding segments of DNA that play a central role in
regulating transcriptional programs that control development, cell identity, and evolutionary …

Genome-wide association study identifies 25 known breast cancer susceptibility loci as risk factors for triple-negative breast cancer

KS Purrington, S Slager, D Eccles… - …, 2014 - academic.oup.com
Triple-negative (TN) breast cancer is an aggressive subtype of breast cancer associated
with a unique set of epidemiologic and genetic risk factors. We conducted a two-stage …

Comprehensive functional annotation of 77 prostate cancer risk loci

DJ Hazelett, SK Rhie, M Gaddis, C Yan… - PLoS …, 2014 - journals.plos.org
Genome-wide association studies (GWAS) have revolutionized the field of cancer genetics,
but the causal links between increased genetic risk and onset/progression of disease …

CRISPR-mediated deletion of prostate cancer risk-associated CTCF loop anchors identifies repressive chromatin loops

Y Guo, AA Perez, DJ Hazelett, GA Coetzee, SK Rhie… - Genome biology, 2018 - Springer
Background Recent genome-wide association studies (GWAS) have identified more than
100 loci associated with increased risk of prostate cancer, most of which are in non-coding …

Using 3D epigenomic maps of primary olfactory neuronal cells from living individuals to understand gene regulation

SK Rhie, S Schreiner, H Witt, C Armoskus, FD Lay… - Science …, 2018 - science.org
As part of PsychENCODE, we developed a three-dimensional (3D) epigenomic map of
primary cultured neuronal cells derived from olfactory neuroepithelium (CNON). We mapped …

Molecular mechanisms underlying noncoding risk variations in psychiatric genetic studies

X **ao, H Chang, M Li - Molecular psychiatry, 2017 - nature.com
Recent large-scale genetic approaches such as genome-wide association studies have
allowed the identification of common genetic variations that contribute to risk architectures of …

Assessing Associations between the AURKA-HMMR-TPX2-TUBG1 Functional Module and Breast Cancer Risk in BRCA1/2 Mutation Carriers

I Blanco, K Kuchenbaecker, D Cuadras, X Wang… - PLoS …, 2015 - journals.plos.org
While interplay between BRCA1 and AURKA-RHAMM-TPX2-TUBG1 regulates mammary
epithelial polarization, common genetic variation in HMMR (gene product RHAMM) may be …

LEM-3 is a midbody-tethered DNA nuclease that resolves chromatin bridges during late mitosis

Y Hong, R Sonneville, B Wang, V Scheidt… - Nature …, 2018 - nature.com
Faithful chromosome segregation and genome maintenance requires the removal of all
DNA bridges that physically link chromosomes before cells divide. Using C. elegans …

Identification of breast cancer associated variants that modulate transcription factor binding

Y Liu, NM Walavalkar, MG Dozmorov, SS Rich… - PLoS …, 2017 - journals.plos.org
Genome-wide association studies (GWAS) have discovered thousands loci associated with
disease risk and quantitative traits, yet most of the variants responsible for risk remain …