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Inflammation after intracerebral hemorrhage
J Wang, S Doré - Journal of Cerebral Blood Flow & …, 2007 - journals.sagepub.com
Intracerebral hemorrhage (ICH) is a devastating clinical event without effective therapies.
Increasing evidence suggests that inflammatory mechanisms are involved in the …
Increasing evidence suggests that inflammatory mechanisms are involved in the …
Potential markers of oxidative stress in stroke
Free radical production is increased in ischemic and hemorrhagic stroke, leading to
oxidative stress that contributes to brain damage. The measurement of oxidative stress in …
oxidative stress that contributes to brain damage. The measurement of oxidative stress in …
BBB leakage, astrogliosis, and tissue loss correlate with silicon microelectrode array recording performance
The clinical usefulness of brain machine interfaces that employ penetrating silicon
microelectrode arrays is limited by inconsistent performance at chronic time points. While it …
microelectrode arrays is limited by inconsistent performance at chronic time points. While it …
Matrix metalloproteinase—9 concentration after spontaneous intracerebral hemorrhage
Object. Matrix metalloproteinases (MMPs) are overexpressed in the presence of some
neurological diseases in which blood—brain barrier disruption exists. The authors …
neurological diseases in which blood—brain barrier disruption exists. The authors …
Brain edema after intracerebral hemorrhage: mechanisms, treatment options, management strategies, and operative indications
✓ Primary intracerebral hemorrhage (ICH) is associated with a high mortality rate and
severe morbidity. The treatment of choice is still controversial, given that data from several …
severe morbidity. The treatment of choice is still controversial, given that data from several …
15d-Prostaglandin J2 Activates Peroxisome Proliferator-Activated Receptor-γ, Promotes Expression of Catalase, and Reduces Inflammation, Behavioral Dysfunction …
Peroxisome proliferator-activated receptor-γ (PPARγ) is a transcription factor that regulates
the expression of various gene products that are essential in lipid and glucose metabolism …
the expression of various gene products that are essential in lipid and glucose metabolism …
Human albumin attenuates excessive innate immunity via inhibition of microglial Mincle/Syk signaling in subarachnoid hemorrhage
Y **e, H Guo, L Wang, L Xu, X Zhang, L Yu… - Brain, behavior, and …, 2017 - Elsevier
Subarachnoid hemorrhage (SAH) is a devastating subtype of stroke. Microglial macrophage-
inducible C-type lectin (Mincle) receptor launches microglial innate immunity after SAH, and …
inducible C-type lectin (Mincle) receptor launches microglial innate immunity after SAH, and …
Acute tissue damage after injections of thrombin and plasmin into rat striatum
Background and Purpose—Extravasation of blood is associated with intracerebral
hemorrhage and head trauma. The mechanism of brain cell injury associated with …
hemorrhage and head trauma. The mechanism of brain cell injury associated with …
Attenuation of intracerebral hemorrhage and thrombin-induced brain edema by overexpression of interleukin-1 receptor antagonist
Object. Adenovirus-mediated overexpression of interleukin-1 receptor antagonist (IL-1ra)
attenuates the inflammatory reaction and brain injury that follows focal cerebral ischemia …
attenuates the inflammatory reaction and brain injury that follows focal cerebral ischemia …
Reduction of neutrophil activity decreases early microvascular injury after subarachnoid haemorrhage
V Friedrich, R Flores, A Muller, W Bi… - Journal of …, 2011 - Springer
Background Subarachnoid haemorrhage (SAH) elicits rapid pathological changes in the
structure and function of parenchymal vessels (≤ 100 μm). The role of neutrophils in these …
structure and function of parenchymal vessels (≤ 100 μm). The role of neutrophils in these …