Stroke induces a plastic state in the brain. This period of enhanced plasticity leads to the
sprouting of new axons, the formation of new synapses and the remap** of sensory-motor …

Since Cajal's first drawings of Golgi stained neurons, generations of researchers have been
fascinated by the small protrusions, termed spines, studding many neuronal dendrites. Most …

We tested a newly described molecular memory system, CCR5 signaling, for its role in
recovery after stroke and traumatic brain injury (TBI). CCR5 is uniquely expressed in cortical …

Stroke remains a leading cause of death and disability in the world. Over the past few
decades our understanding of the pathophysiology of stroke has increased, but greater …

The past 20 years have represented an important period in the development of principles
underlying neuroplasticity, especially as they apply to recovery from neurological injury. It is …

Reductions in blood flow to the brain of sufficient duration and extent lead to stroke, which
results in damage to neuronal networks and the impairment of sensation, movement or …

Ischemic damage to the brain triggers substantial reorganization of spared areas and
pathways, which is associated with limited, spontaneous restoration of function. A better …

Synaptic plasticity in adult neural circuits may involve the strengthening or weakening of
existing synapses as well as structural plasticity, including synapse formation and …

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Brain injury causes astrocytes to assume a reactive state that is essential for early tissue
protection, but how reactive astrocytes affect later reparative processes is incompletely …