In Alzheimer's disease (AD), early synaptic dysfunction is associated with the increased
oligomeric amyloid-beta peptide, which causes NMDAR-dependent synaptic depression …

Modern research has revealed that dietary consumption of flavonoids and flavonoids-rich
foods significantly improve cognitive capabilities, inhibit or delay the senescence process …

Aging and neurodegenerative diseases share common hallmarks, including mitochondrial
dysfunction and protein aggregation. Moreover, one of the major issues of the demographic …

Amyloid plaques and neurofibrillary tangles (NFTs) in the brain are the neuropathological
hallmarks of Alzheimer's disease (AD). Amyloid plaques are composed of β‐amyloid …

Particulate ligands, including cholesterol crystals and amyloid fibrils, induce production of
interleukin 1β (IL-1β) dependent on the cytoplasmic sensor NLRP3 in atherosclerosis …

The amyloid β peptide (Aβ) is a critical initiator that triggers the progression of Alzheimer's
Disease (AD) via accumulation and aggregation, of which the process may be caused by Aβ …

Importance Increasing evidence suggests a relationship between poor sleep and the risk of
develo** Alzheimer disease. A previous study found an effect of sleep on β-amyloid (Aβ) …

The primal role that the amyloid-β (Aβ) peptide has in the development of Alzheimer's
disease is now almost universally accepted. It is also well recognized that Aβ exists in …

Although the exact etiology of Alzheimer's disease (AD) is a topic of debate, the consensus
is that the accumulation of β-amyloid (Aβ) peptides in the senile plaques is one of the …

An important pathological feature of Alzheimer's disease (AD) is the presence of
extracellular senile plaques in the brain. Senile plaques are composed of aggregations of …