SARS-CoV-2 ORF6 disrupts innate immune signalling by inhibiting cellular mRNA export

R Hall, A Guedán, MW Yap, GR Young… - PLoS …, 2022‏ - journals.plos.org
SARS-CoV-2 is a betacoronavirus and the etiological agent of COVID-19, a devastating
infectious disease. Due to its far-reaching effect on human health, there is an urgent and …

Nanoscopic elucidation of spontaneous self-assembly of Severe Acute Respiratory Syndrome Coronavirus 2 (SARS-CoV-2) open reading frame 6 (ORF6) protein

G Nishide, K Lim, M Tamura, A Kobayashi… - The Journal of …, 2023‏ - ACS Publications
Open reading frame 6 (ORF6), the accessory protein of severe acute respiratory syndrome
coronavirus 2 (SARS-CoV-2) that suppresses host type-I interferon signaling, possesses …

AThe M-motif: a potential new class of non-conventional NLS in TAZ/YAP and other cellular and viral proteins that inhibits classic protein import

M Kofler, S Venugopal, G Gill, C Di Ciano-Oliveira… - iScience, 2025‏ - cell.com
Multiple mechanisms were proposed to mediate the nuclear import of TAZ/YAP,
transcriptional co-activators regulating organ growth and regeneration. Our earlier …

Interferon-γ as a Potential Inhibitor of SARS-CoV-2 ORF6 Accessory Protein

E Krachmarova, P Petkov, E Lilkova… - International Journal of …, 2024‏ - mdpi.com
The ORF6 protein of the SARS-CoV-2 virus plays a crucial role in blocking the innate
immune response of the infected cells by inhibiting interferon pathways. Additionally, it binds …

SARS-CoV-2 Orf6 requires the Nup98/Rae1 complex to inhibit host nuclear transport

ND Love - 2023‏ - era.library.ualberta.ca
The nucleus is a hallmark organelle of eukaryotes. It separates the genetic material from the
rest of the cytoplasm using a selectively permeable double phospholipid bilayer called the …