Non-homologous DNA end joining (NHEJ) is the predominant repair mechanism of any type
of DNA double-strand break (DSB) during most of the cell cycle and is essential for the …

In the last two decades, an increasing amount of studies have investigated the use of
components based on carbon-(nano) materials in the engineering of neural interfaces, to …

Double-strand breaks (DSBs) are extremely detrimental DNA lesions that can lead to cancer-
driving mutations and translocations. Non-homologous end joining (NHEJ) and homologous …

DNA double-strand breaks (DSBs) are a highly cytotoxic form of DNA damage and the
incorrect repair of DSBs is linked to carcinogenesis,. The conserved error-prone non …

DNA double-strand breaks pose a serious threat to genome stability. In vertebrates, these
breaks are predominantly repaired by nonhomologous end joining (NHEJ), which pairs DNA …

Nonhomologous end joining (NHEJ) refers to a set of genome maintenance pathways in
which two DNA double-strand break (DSB) ends are (re) joined by apposition, processing …

DNA double-strand breaks (DSBs) are accidental lesions generated by various endogenous
or exogenous stresses. DSBs are also genetically programmed events during the V (D) J …

Non-homologous end joining (NHEJ) is one of two critical mechanisms utilized in humans to
repair DNA double-strand breaks (DSBs). Unrepaired or incorrect repair of DSBs can lead to …

Non-homologous end joining (NHEJ) is the primary pathway for repairing DNA double-
strand breaks (DSBs) in mammalian cells. Such breaks are formed, for example, during …

A DNA double-strand break (DSB) has long been recognized as a severe cellular lesion,
potentially representing an initiating event for carcinogenesis or cell death. The evolution of …