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[HTML][HTML] Mitochondrial pharmacology
Mitochondria are being recognized as key factors in many unexpected areas of biomedical
science. In addition to their well-known roles in oxidative phosphorylation and metabolism, it …
science. In addition to their well-known roles in oxidative phosphorylation and metabolism, it …
Emerging therapies for mitochondrial diseases
For the vast majority of patients with mitochondrial diseases, only supportive and
symptomatic therapies are available. However, in the last decade, due to extraordinary …
symptomatic therapies are available. However, in the last decade, due to extraordinary …
Mitochondrial ROS produced via reverse electron transport extend animal lifespan
Increased production of reactive oxygen species (ROS) has long been considered a cause
of aging. However, recent studies have implicated ROS as essential secondary messengers …
of aging. However, recent studies have implicated ROS as essential secondary messengers …
[HTML][HTML] Oxidative metabolism drives immortalization of neural stem cells during tumorigenesis
Metabolic reprogramming is a key feature of many cancers, but how and when it contributes
to tumorigenesis remains unclear. Here we demonstrate that metabolic reprogramming …
to tumorigenesis remains unclear. Here we demonstrate that metabolic reprogramming …
Mitochondrial complex I activity and NAD+/NADH balance regulate breast cancer progression
Despite advances in clinical therapy, metastasis remains the leading cause of death in
breast cancer patients. Mutations in mitochondrial DNA, including those affecting complex I …
breast cancer patients. Mutations in mitochondrial DNA, including those affecting complex I …
Superoxide is produced by the reduced flavin in mitochondrial complex I: a single, unified mechanism that applies during both forward and reverse electron transfer
NADH ubiquinone oxidoreductase (complex I) is a major source of reactive oxygen species
in mitochondria and a contributor to cellular oxidative stress. In isolated complex I the …
in mitochondria and a contributor to cellular oxidative stress. In isolated complex I the …
Ndufs4 knockout mouse models of Leigh syndrome: pathophysiology and intervention
Mitochondria are small cellular constituents that generate cellular energy (ATP) by oxidative
phosphorylation (OXPHOS). Dysfunction of these organelles is linked to a heterogeneous …
phosphorylation (OXPHOS). Dysfunction of these organelles is linked to a heterogeneous …
Branched-chain amino acids, mitochondrial biogenesis, and healthspan: an evolutionary perspective
Malnutrition is common among older persons, with important consequences increasing
frailty and morbidity and reducing health expectancy. On the contrary, calorie restriction (CR …
frailty and morbidity and reducing health expectancy. On the contrary, calorie restriction (CR …
The advanced lipoxidation end-product malondialdehyde-lysine in aging and longevity
The nonenzymatic adduction of malondialdehyde (MDA) to the protein amino groups leads
to the formation of malondialdehyde-lysine (MDALys). The degree of unsaturation of …
to the formation of malondialdehyde-lysine (MDALys). The degree of unsaturation of …
[HTML][HTML] Physiologic implications of reactive oxygen species production by mitochondrial complex I reverse electron transport
Mitochondrial reactive oxygen species (ROS) can be either detrimental or beneficial
depending on the amount, duration, and location of their production. Mitochondrial complex I …
depending on the amount, duration, and location of their production. Mitochondrial complex I …