Exported proteins of the malaria parasite Plasmodium falciparum interact with proteins of the
erythrocyte membrane and induce substantial changes in the morphology, physiology and …

Plasmodium parasites, the causative agents of malaria, have developed elaborate
strategies that they use to survive and thrive within different intracellular environments …

Several hundred malaria parasite proteins are exported beyond an encasing vacuole and
into the cytosol of the host erythrocyte, a process that is central to the virulence and viability …

During the blood stages of malaria, several hundred parasite-encoded proteins are exported
beyond the double-membrane barrier that separates the parasite from the host cell …

Plasmodium falciparum causes the virulent form of malaria and disease manifestations are
linked to growth inside infected erythrocytes. To survive and evade host responses the …

During their intraerythrocytic development, malaria parasites export hundreds of proteins to
remodel their host cell. Nutrient acquisition, cytoadherence and antigenic variation are …

The hemoglobins S and C protect carriers from severe Plasmodium falciparum malaria.
Here, we found that these hemoglobinopathies affected the trafficking system that directs …

The root knot nematode, Meloidogyne incognita, is an obligate parasite that causes
significant damage to a broad range of host plants. Infection is associated with secretion of …

Microsporidia are a large group of fungal‐related obligate intracellular parasites. They are
responsible for infections in humans as well as in agriculturally and environmentally …

Malaria blood stage parasites export a large number of proteins into their host erythrocyte to
change it from a container of predominantly hemoglobin optimized for the transport of …