Characterization of Pediatric Acute Myeloid Leukemia With t (7; 12)(q36; p13)
Acute myeloid leukemia (AML) with t (7; 12)(q36; p13) is a recurrent translocation in AML in
infants or very young children and was recently included in the World Health Organization …
infants or very young children and was recently included in the World Health Organization …
Identification of gene signatures relevant to the efficacy of immune checkpoint inhibitors in non-small cell lung cancer
M Liu, Q Li, X Meng, Y Cui, W Sun, H Wang, Q Gao - Medicine, 2024 - journals.lww.com
Despite significant advancements in the treatment of non-small cell lung cancer (NSCLC)
through immunotherapy, many patients still exhibit resistance to this approach. This study …
through immunotherapy, many patients still exhibit resistance to this approach. This study …
Gene Expression Profiling to Predict Prognostic Biomarkers for Relapse in Multiple Myeloma
S Elahi, S Fazal - Advancements in Life Sciences, 2024 - als-journal.com
Background: Multiple myeloma (MM), an incurable malignancy of plasma cells (PCs), is the
second most common hematological cancer caused primarily by structural variations …
second most common hematological cancer caused primarily by structural variations …
[BOOK][B] The Translocation t (7; 12)(q36; p13) in Childhood Acute Myeloid Leukemia
A Östlund - 2024 - gupea.ub.gu.se
The reciprocal translocation t (7; 12)(q36; p13) gives rise to acute myeloid leukemia (AML) in
infants and very young children. A fusion transcript MNX1:: ETV6 is sometimes detected and …
infants and very young children. A fusion transcript MNX1:: ETV6 is sometimes detected and …
Epigenetic compound screening in AML with MNX1 overexpression
S Kelekçi - 2024 - archiv.ub.uni-heidelberg.de
Acute myeloid leukemia (AML) is a form of blood cancer characterized by impaired
differentiation and accelerated proliferation due to (epi) genetic dysregulation of …
differentiation and accelerated proliferation due to (epi) genetic dysregulation of …