Tumor suppressive role for kinases phosphorylating p53 in DNA damage‐induced apoptosis

S Yogosawa, K Yoshida - Cancer science, 2018 - Wiley Online Library
Tumor suppressor p53 plays an important role in cancer prevention. Under normal
conditions, p53 is maintained at a low level. However, in response to various cellular …

The DYRK family of kinases in cancer: molecular functions and therapeutic opportunities

J Boni, C Rubio-Perez, N López-Bigas, C Fillat… - Cancers, 2020 - mdpi.com
DYRK (dual-specificity tyrosine-regulated kinases) are an evolutionary conserved family of
protein kinases with members from yeast to humans. In humans, DYRKs are pleiotropic …

Site-specific proteasome phosphorylation controls cell proliferation and tumorigenesis

X Guo, X Wang, Z Wang, S Banerjee, J Yang… - Nature cell …, 2016 - nature.com
Despite the fundamental importance of proteasomal degradation in cells, little is known
about whether and how the 26S proteasome itself is regulated in coordination with various …

FBXW7 tumor suppressor regulation by dualspecificity tyrosine-regulated kinase 2

R Jiménez-Izquierdo, R Morrugares… - Cell Death & …, 2023 - nature.com
FBXW7 is a member of the F-box protein family, which functions as the substrate recognition
component of the SCF E3 ubiquitin ligase. FBXW7 is a main tumor suppressor due to its …

Emerging roles of DYRK2 in cancer

V Tandon, L de la Vega, S Banerjee - Journal of Biological Chemistry, 2021 - ASBMB
Over the last decade, the CMGC kinase DYRK2 has been reported as a tumor suppressor
across various cancers triggering major antitumor and proapoptotic signals in breast, colon …

Structures of Down syndrome kinases, DYRKs, reveal mechanisms of kinase activation and substrate recognition

M Soundararajan, AK Roos, P Savitsky… - Structure, 2013 - cell.com
Summary Dual-specificity tyrosine-(Y)-phosphorylation-regulated kinases (DYRKs) play key
roles in brain development, regulation of splicing, and apoptosis, and are potential drug …

[HTML][HTML] Genome-wide profiling identifies a DNA methylation signature that associates with TET2 mutations in diffuse large B-cell lymphoma

F Asmar, V Punj, J Christensen, MT Pedersen… - …, 2013 - ncbi.nlm.nih.gov
The discovery that the Ten-Eleven Translocation (TET) hydroxylases cause DNA
demethylation has fundamentally changed the notion of how DNA methylation is regulated …

A broad activity screen in support of a chemogenomic map for kinase signalling research and drug discovery

Y Gao, SP Davies, M Augustin, A Woodward… - Biochemical …, 2013 - portlandpress.com
Despite the development of a number of efficacious kinase inhibitors, the strategies for
rational design of these compounds have been limited by target promiscuity. In an effort to …

An overview of cdc2‐like kinase 1 (Clk1) inhibitors and their therapeutic indications

AK ElHady, DS El‐Gamil, AH Abadi… - Medicinal Research …, 2023 - Wiley Online Library
Over the past decade, Clk1 has been identified as a promising target for the treatment of
various diseases, in which deregulated alternative splicing plays a role. First small …

A novel CDC25A/DYRK2 regulatory switch modulates cell cycle and survival

M Lara-Chica, A Correa-Sáez… - Cell Death & …, 2022 - nature.com
The cell division cycle 25A (CDC25A) phosphatase is a key regulator of cell cycle
progression that acts on the phosphorylation status of Cyclin–Cyclin-dependent kinase …