Exposure to pro-inflammatory cytokines, chemokines, mitochondrial contents, and bacterial
and viral products induces neutrophils to transition from a basal state into a primed one …

2. Tachykinins and CGRP in primary afferent neurons 2.1. Synthesis and expression of
tachykinins 2.2. Svnthesis and exnression of CGRP 2.3. Co-localization of tachykinins and …

Nervous connections between the trigeminal ganglia and cerebral blood vessels have
recently been identified in experimental animals and have been termed the …

A smoking-induced inflammatory reaction in the airways and lung parenchyma, comprised
mainly of neutrophils and alveolar macrophages, has long been accepted to be the major …

Several clinical features are consistent with nervous system involvement in the
pathogenesis of rheumatoid arthritis. The neuropeptide substance P is one possible …

The role of stress as a triggering factor in the exacerbation of psoriasis and the clinically
symmetric distribution of psoriatic plaques suggested a possible role for neuropeptides in …

Gingivitis and periodontitis are thought to result from an imbalance between those oral
microorganisms which normally colonize tooth surfaces in close contact with the gingival …

There is evidence that substance P is a peptide neurotransmitter of some unmyelinated
primary afferent nociceptors and that its release from the peripheral terminals of primary …

The aim of this article is to provide an up-dated overview of the available information on the
role played by tachykinins in recruiting/regulating the function of immune/inflammatory cells …

Neurogenic stimuli profoundly affect cellular events that are involved in inflammation,
proliferation, and matrix, as well as cytokine and growth factor synthesis. Immune cells …