Amyloid oligomers: A joint experimental/computational perspective on Alzheimer's disease, Parkinson's disease, type II diabetes, and amyotrophic lateral sclerosis

PH Nguyen, A Ramamoorthy, BR Sahoo… - Chemical …, 2021 - ACS Publications
Protein misfolding and aggregation is observed in many amyloidogenic diseases affecting
either the central nervous system or a variety of peripheral tissues. Structural and dynamic …

Tetrameric Aβ40 and Aβ42 β-barrel structures by extensive atomistic simulations. II. In aqueous solution

PH Nguyen, JM Campanera, ST Ngo… - The Journal of …, 2019 - ACS Publications
Alzheimer's disease (AD) is characterized by the accumulation of extracellular Aβ42 and
Aβ40 oligomers and plaques. In a recent computational study, we found that the presence of …

Effect of Terahertz Waves on the Structure of the Aβ42 Monomer, Dimer, and Protofibril: Insights from Molecular Dynamics Simulations

C Chen, ZS Yan, YQ Ma, HM Ding - ACS Chemical Neuroscience, 2023 - ACS Publications
Amyloid-β (Aβ) and its assemblies play important roles in the pathogenesis of Alzheimer's
disease (AD). Recent studies conducted by experimental and computational researchers …

Molecular dynamics simulations reveal the mechanism of graphene oxide nanosheet inhibition of Aβ 1–42 peptide aggregation

Y **, Y Sun, Y Chen, J Lei, G Wei - Physical Chemistry Chemical …, 2019 - pubs.rsc.org
The aggregation of the amyloid-beta (Aβ) peptides into toxic β-sheet-rich oligomers,
protofibrils and mature fibrils is the major pathological hallmark of Alzheimer's disease (AD) …

Mechanistic insights into the mitigation of Aβ aggregation and protofibril destabilization by ad-enantiomeric decapeptide rk10

K Singh, A Kaur, D Goyal, B Goyal - Physical Chemistry Chemical …, 2022 - pubs.rsc.org
According to clinical studies, the development of Alzheimer's disease (AD) is linked to the
abnormal aggregation of amyloid-β (Aβ) peptides into toxic soluble oligomers, protofibrils as …

Amyloid β dodecamer disrupts the neuronal membrane more strongly than the mature fibril: Understanding the role of oligomers in neurotoxicity

HL Nguyen, HQ Linh, P Krupa… - The Journal of …, 2022 - ACS Publications
The amyloid cascade hypothesis states that senile plaques, composed of amyloid β (Aβ)
fibrils, play a key role in Alzheimer's disease (AD). However, recent experiments have …

The role of structural polymorphism in driving the mechanical performance of the alzheimer's beta amyloid fibrils

G Grasso, M Rebella, U Morbiducci… - … in bioengineering and …, 2019 - frontiersin.org
Alzheimer's Disease (AD) is related with the abnormal aggregation of amyloid β-peptides
Aβ1− 40 and Aβ1− 42, the latter having a polymorphic character which gives rise to U-or S …

Emergence of barrel motif in amyloid-β trimer: a computational study

HL Nguyen, HQ Linh, P Matteini… - The Journal of …, 2020 - ACS Publications
Amyloid-β (Aβ) peptides form assemblies that are pathological hallmarks of Alzheimer's
disease. Aβ oligomers are soluble, mobile, and toxic forms of the peptide that act in the …

Structures and Dynamics of β-Rich Oligomers of ATTR (105–115) Assembly

L Liang, Y Zhang, Y Zhu, J Bai, Y Ni… - ACS Chemical …, 2024 - ACS Publications
Transthyretin (TTR) is a tetrameric homologous protein that can dissociate into monomers.
Misfolding and aggregation of TTR can lead to amyloid transthyretin amyloidosis (ATTR) …

Distinguishing the effect on the rate and yield of Aβ42 aggregation by green tea polyphenol EGCG

G Park, C Xue, H Wang, Z Guo - ACS omega, 2020 - ACS Publications
Deposition of Aβ42 aggregates in the form of amyloid plaques is a pathological hallmark of
Alzheimer's disease. A desired avenue of intervention is the inhibition of Aβ42 aggregation …