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Functions and mechanisms of non-histone protein acetylation
N ε-lysine acetylation was discovered more than half a century ago as a post-translational
modification of histones and has been extensively studied in the context of transcription …
modification of histones and has been extensively studied in the context of transcription …
NAD+ Metabolism in Cardiac Health, Aging, and Disease
Nicotinamide adenine dinucleotide (NAD+) is a central metabolite involved in energy and
redox homeostasis as well as in DNA repair and protein deacetylation reactions …
redox homeostasis as well as in DNA repair and protein deacetylation reactions …
[HTML][HTML] Mitochondrial-targeted therapy for doxorubicin-induced cardiotoxicity
Anthracyclines, such as doxorubicin, are effective chemotherapeutic agents for the treatment
of cancer, but their clinical use is associated with severe and potentially life-threatening …
of cancer, but their clinical use is associated with severe and potentially life-threatening …
Sirtuins and NAD+ in the Development and Treatment of Metabolic and Cardiovascular Diseases
The sirtuin family of nicotinamide adenine dinucleotide–dependent deacylases (SIRT1–7)
are thought to be responsible, in large part, for the cardiometabolic benefits of lean diets and …
are thought to be responsible, in large part, for the cardiometabolic benefits of lean diets and …
SIRT1 and SIRT6 signaling pathways in cardiovascular disease protection
Significance: Oxidative stress represents the common hallmark of pathological conditions
associated with cardiovascular disease (CVD), including atherosclerosis, heart failure …
associated with cardiovascular disease (CVD), including atherosclerosis, heart failure …
Lysine acetyltransferases and lysine deacetylases as targets for cardiovascular disease
P Li, J Ge, H Li - Nature Reviews Cardiology, 2020 - nature.com
Lysine acetylation is a conserved, reversible, post-translational protein modification
regulated by lysine acetyltransferases (KATs) and lysine deacetylases (KDACs; also known …
regulated by lysine acetyltransferases (KATs) and lysine deacetylases (KDACs; also known …
Tumor cells impair immunological synapse formation via central nervous system-enriched metabolite
Y Li, M Huang, M Wang, Y Wang, P Deng, C Li… - Cancer Cell, 2024 - cell.com
Tumors employ various strategies to evade immune surveillance. Central nervous system
(CNS) has multiple features to restrain immune response. Whether tumors and CNS share …
(CNS) has multiple features to restrain immune response. Whether tumors and CNS share …
Dapagliflozin attenuates pressure overload-induced myocardial remodeling in mice via activating SIRT1 and inhibiting endoplasmic reticulum stress
F Ren, Z **e, Y Jiang, X Guan, Q Chen… - Acta Pharmacologica …, 2022 - nature.com
Endoplasmic reticulum stress-mediated apoptosis plays a vital role in the occurrence and
development of heart failure. Dapagliflozin (DAPA), a new type of sodium-glucose …
development of heart failure. Dapagliflozin (DAPA), a new type of sodium-glucose …
Autophagy-dependent and-independent modulation of oxidative and organellar stress in the diabetic heart by glucose-lowering drugs
Autophagy is a lysosome-dependent intracellular degradative pathway, which mediates the
cellular adaptation to nutrient and oxygen depletion as well as to oxidative and endoplasmic …
cellular adaptation to nutrient and oxygen depletion as well as to oxidative and endoplasmic …
[HTML][HTML] Energy metabolism disorders and potential therapeutic drugs in heart failure
Y He, W Huang, C Zhang, L Chen, R Xu, N Li… - … Pharmaceutica Sinica B, 2021 - Elsevier
Heart failure (HF) is a global public health problem with high morbidity and mortality. A large
number of studies have shown that HF is caused by severe energy metabolism disorders …
number of studies have shown that HF is caused by severe energy metabolism disorders …