Signaling pathways in cancer: therapeutic targets, combinatorial treatments, and new developments

HYK Yip, A Papa - Cells, 2021 - mdpi.com
Molecular alterations in cancer genes and associated signaling pathways are used to inform
new treatments for precision medicine in cancer. Small molecule inhibitors and monoclonal …

Resistance to CDK4/6 inhibitors in estrogen receptor-positive breast cancer

ER Scheidemann, AN Shajahan-Haq - International Journal of Molecular …, 2021 - mdpi.com
Estrogen receptor-positive (ER+) breast cancer is the most common form of breast cancer.
Antiestrogens were the first therapy aimed at treating this subtype, but resistance to these …

A deep learning model of tumor cell architecture elucidates response and resistance to CDK4/6 inhibitors

S Park, E Silva, A Singhal, MR Kelly, K Licon… - Nature cancer, 2024 - nature.com
Cyclin-dependent kinase 4 and 6 inhibitors (CDK4/6is) have revolutionized breast cancer
therapy. However,< 50% of patients have an objective response, and nearly all patients …

[HTML][HTML] Expanding control of the tumor cell cycle with a CDK2/4/6 inhibitor

K Freeman-Cook, RL Hoffman, N Miller, J Almaden… - Cancer cell, 2021 - cell.com
Summary The CDK4/6 inhibitor, palbociclib (PAL), significantly improves progression-free
survival in HR+/HER2− breast cancer when combined with anti-hormonals. We sought to …

Co-targeting CDK2 and CDK4/6 overcomes resistance to aromatase and CDK4/6 inhibitors in ER+ breast cancer

AJ Al-Qasem, CL Alves, S Ehmsen… - Npj Precision …, 2022 - nature.com
Resistance to aromatase inhibitor (AI) treatment and combined CDK4/6 inhibitor (CDK4/6i)
and endocrine therapy (ET) are crucial clinical challenges in treating estrogen receptor …

Seize the engine: Emerging cell cycle targets in breast cancer

J Fuentes‐Antrás, PL Bedard… - Clinical and …, 2024 - Wiley Online Library
Breast cancer arises from a series of molecular alterations that disrupt cell cycle
checkpoints, leading to aberrant cell proliferation and genomic instability. Targeted …

[HTML][HTML] EGFR and HER2 hyper-activation mediates resistance to endocrine therapy and CDK4/6 inhibitors in ER+ breast cancer

S Belli, D Esposito, CM Ascione, F Messina… - Cancer letters, 2024 - Elsevier
In patients with ER+ metastatic breast cancer (mBC), the first-line treatment involves the
combination of endocrine therapy (ET) and CDK4/6 inhibitors (CDK4/6i). However, a …

Functional activation of the AKT-mTOR signalling axis in a real-world metastatic breast cancer cohort

D Prasad, E Baldelli, EM Blais, J Davis… - British Journal of …, 2024 - nature.com
Background Mutations of the PIK3CA/AKT/mTOR axis are common events in metastatic
breast cancers (MBCs). This study was designed to evaluate the extent to which genetic …

[HTML][HTML] CDK4/6 inhibitor resistance in hormone receptor-positive metastatic breast cancer: translational research, clinical trials, and future directions

JS Lee, H Hackbart, X Cui, Y Yuan - International Journal of Molecular …, 2023 - mdpi.com
The emergence of CDK4/6 inhibitors, such as palbociclib, ribociclib, and abemaciclib, has
revolutionized the treatment landscape for hormone receptor-positive breast cancer. These …