Reperfusion is mandatory to salvage ischemic myocardium from infarction, but reperfusion
per se contributes to injury and ultimate infarct size. Therefore, cardioprotection beyond that …

Ischaemic postconditioning (brief periods of ischaemia alternating with brief periods of
reflow applied at the onset of reperfusion following sustained ischaemia) effectively reduces …

Reperfusion therapy must be applied as soon as possible to attenuate the ischemic insult of
acute myocardial infarction (AMI). However reperfusion is responsible for additional …

Over the past 2 to 3 decades, several phenomena have been identified that provide
powerful protection against myocardial infarction and other sequelae of …

Nitric oxide (NO) participates in the control of contractility and heart rate, limits cardiac
remodeling after an infarction and contributes to the protective effect of ischemic pre-and …

Abstract Post–conditioning (Post–C) induced cardioprotection involves activation of guanylyl–
cyclase. In the ischemic preconditioning scenario, the downstream targets of cGMP include …

A series of brief (a few minutes) ischemia/reperfusion cycles (ischemic preconditioning, IP)
limits myocardial injury produced by a subsequent prolonged period of coronary artery …

Ischemic postconditioning, a simple mechanical maneuver at the onset of reperfusion,
reduces infarct size after ischemia/reperfusion. After its first description in 2003 by Zhao et al …

Reperfusion therapy is the indispensable treatment of acute myocardial infarction (AMI) and
must be applied as soon as possible to attenuate the ischemic insult. However, reperfusion …

Coronary artery disease is a major cause of morbidity and mortality in the Western countries.
Acute myocardial infarction is a serious and often lethal consequence of coronary artery …