Non-coding RNAs in glioma
R Rynkeviciene, J Simiene, E Strainiene… - Cancers, 2018 - mdpi.com
Glioma is the most aggressive brain tumor of the central nervous system. The ability of
glioma cells to migrate, rapidly diffuse and invade normal adjacent tissue, their sustained …
glioma cells to migrate, rapidly diffuse and invade normal adjacent tissue, their sustained …
Non-coding RNAs and glioblastoma: Insight into their roles in metastasis
Glioma, also known as glioblastoma multiforme (GBM), is the most prevalent and most lethal
primary brain tumor in adults. Gliomas are highly invasive tumors with the highest death rate …
primary brain tumor in adults. Gliomas are highly invasive tumors with the highest death rate …
GAS41 modulates ferroptosis by anchoring NRF2 on chromatin
YEATS domain-containing protein GAS41 is a histone reader and oncogene. Here, through
genome-wide CRISPR-Cas9 screenings, we identify GAS41 as a repressor of ferroptosis …
genome-wide CRISPR-Cas9 screenings, we identify GAS41 as a repressor of ferroptosis …
[HTML][HTML] Glioblastoma quo vadis: Will migration and invasiveness reemerge as therapeutic targets?
F Lefranc, E Le Rhun, R Kiss, M Weller - Cancer treatment reviews, 2018 - Elsevier
Purpose The purpose of the current review is to highlight, on one hand, the fact that the
migratory pattern of glioma cells is the major obstacle to combat them with chemotherapy …
migratory pattern of glioma cells is the major obstacle to combat them with chemotherapy …
miRNA signature in glioblastoma: Potential biomarkers and therapeutic targets
O Rezaei, K Honarmand, S Nateghinia, M Taheri… - Experimental and …, 2020 - Elsevier
MicroRNAs (miRNAs) are transcripts with sizes of about 22 nucleotides, which are produced
through a multistep process in the nucleus and cytoplasm. These transcripts modulate the …
through a multistep process in the nucleus and cytoplasm. These transcripts modulate the …
MicroRNA-125a-5p plays a role as a tumor suppressor in lung carcinoma cells by directly targeting STAT3
L Zhong, S Sun, J Shi, F Cao, X Han… - Tumor Biology, 2017 - journals.sagepub.com
Increasing evidence supports that the dysregulation of microRNA expression plays an
important role in the process of tumor occurrence and development. Studies have found that …
important role in the process of tumor occurrence and development. Studies have found that …
MicroRNA-203 suppresses proliferation in liver cancer associated with PIK3CA, p38 MAPK, c-Jun, and GSK3 signaling
A Zhang, J Lakshmanan, A Motameni… - Molecular and cellular …, 2018 - Springer
Primary liver cancer (hepatocellular carcinoma, HCC) is a leading cause of cancer-related
deaths, and alternative ways to treat this disease are urgently needed. In recent years, novel …
deaths, and alternative ways to treat this disease are urgently needed. In recent years, novel …
[HTML][HTML] Bufalin inhibits cellular proliferation and cancer stem cell-like phenotypes via upregulation of MiR-203 in glioma
T Liu, C Wu, G Weng, Z Zhao, X He, C Fu… - Cellular Physiology and …, 2017 - karger.com
Background/Aims: Prior studies have shown that bufalin inhibits cellular proliferation and
induces apoptosis in various human cancers. MicroRNA-203 (miR-203) has been shown to …
induces apoptosis in various human cancers. MicroRNA-203 (miR-203) has been shown to …
Dynamic changes in DICER levels in adipose tissue control metabolic adaptations to exercise
DICER is a key enzyme in microRNA (miRNA) biogenesis. Here we show that aerobic
exercise training up-regulates DICER in adipose tissue of mice and humans. This can be …
exercise training up-regulates DICER in adipose tissue of mice and humans. This can be …
Drug-induced modifications and modulations of microRNAs and long non-coding RNAs for future therapy against Glioblastoma Multiforme
Non-coding RNAs are known to participate in cancer initiation, progression, and metastasis
by regulating the status of chromatin epigenetics and gene expression. Although these non …
by regulating the status of chromatin epigenetics and gene expression. Although these non …