The intestinal hormones glucose-dependent insulinotropic polypeptide (GIP) and glucagon-
like peptide-2 (GLP-2) are key regulators of postprandial bone turnover in humans. We …

To date, no therapy has been proven to be efficacious in fully restoring neurological
functions after spinal cord injury (SCI). Systemic high-dose methylprednisolone (MP) …

Spinal cord injury (SCI) results in marked atrophy of sublesional skeletal muscle and
substantial loss of bone. In this study, the effects of prolonged electrical stimulation (ES) …

The administration of high-dose methylprednisolone (MP) for 24–48 h after traumatic spinal
cord injury (SCI) has been shown to improve functional recovery. The known adverse effects …

Diminished bone perfusion develops in response to disuse and has been proposed as a
mechanism underlying bone loss. Bone blood flow (BF) has not been investigated within the …

Objective Characterise the spatiotemporal responses of trabecular and cortical bone to
complete spinal cord injury (SCI) in the skeletally mature rat in the acute (4-week) period …

Rapid and extensive sublesional bone loss after spinal cord injury (SCI) is a difficult medical
problem that has been refractory to available interventions except the antiresorptive agent …

Objective: Characterise the spatiotemporal trabecular and cortical bone responses to
complete spinal cord injury (SCI) in young rats. Methods: 8-week-old male Wistar rats …

Metabolic acidosis (MA), a disease affecting millions annually, is clinically characterized by
a decrease in systemic pH and bicarbonate. MA has been shown to have skeletal …

spinal cord injury is a devastating neurological disease that causes paralysis of muscles
below the level of damage and causes immobilization. This condition can also cause …