Inflammatory aspects of Alzheimer's disease

P Botella Lucena, MT Heneka - Acta Neuropathologica, 2024 - Springer
Alzheimer´ s disease (AD) stands out as the most common chronic neurodegenerative
disorder. AD is characterized by progressive cognitive decline and memory loss, with …

Microglial function, INPP5D/SHIP1 signaling, and NLRP3 inflammasome activation: implications for Alzheimer's disease

G Terzioglu, TL Young-Pearse - Molecular Neurodegeneration, 2023 - Springer
Recent genetic studies on Alzheimer's disease (AD) have brought microglia under the
spotlight, as loci associated with AD risk are enriched in genes expressed in microglia …

The niche matters: origin, function and fate of cns-associated macrophages during health and disease

A Dalmau Gasull, M Glavan, SKR Samawar… - Acta …, 2024 - Springer
There are several cellular and acellular structural barriers associated with the brain
interfaces, which include the dura, the leptomeninges, the perivascular space and the …

Disease and brain region specific immune response profiles in neurodegenerative diseases with pure and mixed protein pathologies

T Bathe, GP Hery, JAB Villareal, JL Phillips… - Acta Neuropathologica …, 2024 - Springer
The disease-specific accumulation of pathological proteins has long been the major focus of
research in neurodegenerative diseases (ND), including Alzheimer's disease (AD) and …

The role of inhibitory immune checkpoint receptors in the pathogenesis of Alzheimer's disease

A Salminen - Journal of Molecular Medicine, 2024 - Springer
There is mounting evidence that microglial cells have a key role in the pathogenesis of
Alzheimer's disease (AD). In AD pathology, microglial cells not only are unable to remove β …

Microglia promote extracellular matrix deposition and restrict excitatory synapse numbers in the mesolimbic dopamine system during healthy aging

DT Gray, A Guitierrez, Y Jami-Alahmadi, V Pandey… - BioRxiv, 2024 - biorxiv.org
Synapse dysfunction has been definitively linked to cognitive impairments in the aging brain,
and microglial physiology has emerged as a robust regulator of synapse status and …