Jun N-terminal kinase (JNK) and p38 mitogen-activated protein kinase (MAPK) family
members function in a cell context-specific and cell type-specific manner to integrate signals …

c-Jun N-terminal kinases (JNKs), first characterized as stress-activated members of the
mitogen-activated protein kinase (MAPK) family, have become a focus of inhibitor screening …

The cJun NH2-terminal kinase (JNK) signaling pathway contributes to inflammation and
plays a key role in the metabolic response to obesity, including insulin resistance …

The cJun NH 2-terminal kinase (JNK) stress signaling pathway is implicated in the metabolic
response to the consumption of a high-fat diet, including the development of obesity and …

Although alveolar epithelial cells play a critical role in the pathogenesis of pulmonary
fibrosis, few practical in vitro models exist to study them. Here, we established a novel in …

The cJun N-terminal kinase (JNK) signal transduction pathway is implicated in the regulation
of neuronal function. JNK is encoded by three genes that play partially redundant roles …

Genotoxic stress in mammalian cells, including those caused by anti-cancer chemotherapy,
can induce temporary cell-cycle arrest, DNA damage-induced senescence (DDIS), or …

The cJun NH2-terminal kinase (JNK) signal transduction pathway has been implicated in the
growth of carcinogen-induced hepatocellular carcinoma. However, the mechanism that …

c-Jun NH2-terminal kinase (JNK) was discovered almost 20 years ago as the protein kinase
responsible for phosphorylating c-Jun at Ser-63 and Ser-73. These sites had previously …

Abstract Annexin A2 (ANXA2) is widely used as a marker in a variety of tumors. By
regulating multiple signal pathways, ANXA2 promotes the epithelial‐mesenchymal …