In this review, we focus on the potential role of the γ-aminobutyric acidergic (GABAergic)
system in age-related episodic memory impairments in humans, with a particular focus on …

A growing body of evidence has demonstrated a link between Alzheimer disease (AD) and
epilepsy. Late-onset epilepsy and epileptiform activity can precede cognitive deterioration in …

Sepsis-associated encephalopathy (SAE) is a critical neurological complication of sepsis
and represents a crucial factor contributing to high mortality and adverse prognosis in septic …

Alzheimer's disease (AD) is the commonest neurodegenerative disease with slow
progression. Pieces of evidence suggest that the GABAergic system is impaired in the early …

Alzheimer's disease (AD) is a progressive neurodegenerative disease. The accumulation of
amyloid-β (Aβ) plaques and tau neurofibrillary tangles are the key players responsible for …

Abstract Voltage‐gated CaV2. 1 (P/Q‐type) Ca2+ channels play a crucial role in regulating
neurotransmitter release, thus contributing to synaptic plasticity and to processes such as …

Alzheimer's disease (AD) is characterized by a reorganization of brain activity determining
network hyperexcitability and loss of synaptic plasticity. Precisely, a dysfunction in …

Among the central features of Alzheimer's disease (AD) progression are altered levels of the
neuropeptide somatostatin (SST), and the colocalisation of SST-positive interneurons (SST …

Early detection of Alzheimer's disease (AD) is essential for improving the patients outcomes
and advancing our understanding of disease, allowing for timely intervention and treatment …

Synaptic dysfunction is an early feature in Alzheimer's disease (AD) pathogenesis and a
major morphological correlate of memory deficits. Given the main synaptic location of N …