Various factors have been considered as potential seizure precipitants. We here assessed
the temporal association of food intake and seizure occurrence, and characteristics of …

This study aimed to evaluate the clinical, video electroencephalographic and MRI attributes
of patients with eating epilepsy (EE). Consecutive patients who were diagnosed with EE and …

Food intake triggers convulsions in fasted mice and rats treated with antimuscarinic drugs,
scopolamine or atropine. Bearing some similarities in triggering factor and manifestations of …

This study evaluated the effects of different doses of atropine and new antiepileptics,
levetiracetam and topiramate, on the development of convulsions triggered by food intake in …

Antimuscarinic administration and food intake cause convulsions in mice and rats after
fasting for 48 h or less. Increased M 1 and M 2 muscarinic receptor expression in brain …

Background Seizures triggered by eating (STE) behavior are very rare in humans and have
not been documented previously in dogs. Objectives To document the occurrence of STE in …

Abstract Treatment of fasted mice and rats with the nonselective muscarinic antagonist,
scopolamine or atropine, causes convulsions after food intake. This study evaluated the …

Objective: Atropine reduces ghrelin secretion and ghrelin inhibits epileptic seizures. It is
interesting that atropine treated fasting animals develop clonic convulsions soon after food …

Objective (s): Fasted rodents treated with antimuscarinics develop convulsions after
refeeding. Food deprivation for 48 hr produces changes in [3H] glutamate binding …

Objective: Antimuscarinic treatment in fasted mice and rats causes clonic convulsion soon
after food intake. This study was designed to evaluate the electrophysiological markers of …